#Lipid Disorders | Explore Tumblr posts and blogs

brooke121000 · 4 months ago

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Hi! This is a request. Something along the lines of Reader sitting on Spencer Reid’s lap as he talks about his special interests and his hands happen to wander all over your body. Make it as smutty or fluffy as you’d like! Thank you!!

wandering • S. Reid

Make it as smutty or fluffy as you’d like, you say??😈😈 I say both. full disclosure, I did write him a bit more dom then my usual. Ty!

word count: 1185

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Spencer liked to talk. a lot.

It bothered people, very frequently actually- but you on the other hand? You thought it was charming. Spencer liked that about you, you were always willing to listen. No matter what station his train of thought was rolling through that day- you’d be there, head resting in your hands, nodding and commenting, your eyes never leaving his.

This particular day, his mind was set on rare neurological disorders. You were sat in his lap on the couch, fidgeting with the ends of his sleeves while he spoke. A nature documentary was playing on your tv, but neither of you were paying attention, so much so you had turned the volume off.

“Oh, another one-“ he grinned,resting his warm hands on your waist. “Metachromatic leukodystrophy. It’s genetic, actually- autosomal recessive.”

“Oh, what does that one do?” You queried, doing your best not to sound disinterested although you were preoccupied with his fraying sweater seams.

“Basically, our brains and nerves are very delicate. There’s a substance called lipids that build up frequently on the brain, spinal chord and peripheral nerves-“ he interrupted his own sentence to place a small kiss on the crook of your neck, sending warmth down through your collarbones. Despite the loving gesture, you frowned.

“That’s sounds scary.”

“It is, it is. Luckily, we all have enzymes whose sole jobs are to break down those lipids. People with Metachromatic leukodystrophy-“

You interrupted with a guess, raising your head to meet his eyes. “Don’t produce the enzymes?”

He grinned. “Exactly, love.”

“I guessed.”

“Well-“ he shrugged. “It was a good guess.” He pressed a kiss to the corner of your lips, going back to his rant.

“Oddly enough, it actually has similar symptoms to Kuru disease- that’s the one that causes tremors. Only lipids don’t eat away at the tissue,” his hands slowly slid up your sides, caressing your arms and trailing along the curve of your breasts. “..they just block it off.”

You giggled a little at the touch, face warming as you shifted in his lap.

“Stop it.” He said softly, with a smile, guiding your hips down. “It causes a lot of terrible symptoms, anyway. Loss of senses- the most interesting being an inability to detect pain.”

He sneaked another kiss to the side of your neck, a hand moving up to push your hair out of the way.

“It was discovered in the early 20th century, and three forms emerged. Infantile,” his hands slid down your sides, lips pressing quick, sloppy kisses to your jawline.

“juvenile,” as his hands slipped over your thighs, caressing the skin, “and adult.” As his hands expertly pushed your knees apart.

Your breath hitched. “Mhm, spence-“

He sighed through his nose. “Are you listening?”

“yeah, yeah, I am, just- keep going.”

His chin rested on top of your head as he firmly grabbed your hips, spinning you around so your forehead was against his chest, your legs straddling him.

“okay. Pay attention- back to MLD. The infantile form is, of course, the worst, it progresses the quickest and the symptoms are often the most brutal, particularly-“

His hands slid between your thighs, sending a rush up your core, and you pushed your head into his chest a little in a sad attempt of soothing your nerves.

“particularly seizures. Because of this, it’s misdiagnosed often, usually for some form of epilepsy.”

Was he really going to do this? While lecturing you about seizures? Jesus, this boy was going to kill you someday.

His fingers traced along the lace of your panties, one hand situated between the plush of your thighs, one on your waist. The contrast between the movements of his hands and the subject matter leaving his mouth was giving you whiplash, but you were so desperate for any kind of physical attention you let him continue speaking.

“There’s no cure, obviously, there rarely is for anything genetic and neurological.” He spoke, tone never faltering- even as his hand pushed your panties to the side, running along the slick of your folds and pressing a small circle to your clit- causing fireworks to erupt in your core and causing you to let out a long whine, muffled by the fabric of his shirt.

He pinched your side, gently, just as a reminder. “Shhh, love. Don’t you want to hear what I’m saying?”

you managed a shaky nod.

“Good.” He spoke simply, his fingers continuing to work expert circles into you.

“Anyway- before I was interrupted, I was going to say- there’s no cure, but there’s ways to keep the patient comfortable.”

At this point his voice was sure, constant and gentle, causing you to nearly have to strain to hear him correctly. His hands never faltered- and your muscles clenched, thighs tightening around him as the smell of his cologne enclosed your senses.

Your breath was quick, quieted whines and whimpers peaking through, although muffled by the thick material of his sweater. He continued. “Research, mainly in Europe, is proving stem cell treatment to be beneficial.. but that’s mostly in younger patients.”

Your breath hitched when his fingers just slightly trailed down, circling around your entrance and slowly pushing in as his voice persisted.

“a-ah!”

“Shush. You really need to listen, baby.”

His fingers pushed in and out of you, speed never falling even once. “There’s a drug, that was also developed in Europe- called Atidarsagene autotemcel.”

Your hips circled, his fingers curling as he worked you up to the edge. A choked moan escaped your lips, which he ignored.

“baby, cmon- I was saying, since metachromatic leukodystrophy affects the ARSA gene, the treatment takes hematopoietic stem cells from the patient and genetically modifies them to contain a fake, corrected ARSA gene-“

You moaned into his chest, your thighs clenching tighter around him- one hand flattening onto his back for purchase as his ministrations continued, pulling you closer to your inevitable climax.

“g-god, Spencer, can you just- please,”

His movements sped up. “Please, what? I don’t think you even know what you’re asking for, baby. You don’t have to worry, I don’t plan on stopping.”

You were lost for words, breath leaving your chest as his words began to blur together. You babbled something out, your back arching as fireworks lit up your nerves, pleasure washing over you in the midst of your climax.

“s-Spence!”

You pushed your forehead into his chest as his fingers worked you through your orgasm, his free hand coming up to gently hold the back of your head.

“shh, I know,” his hands withdrew from you, slipping out from your thighs and out from under your skirt. He grabbed your chin and allowed your lips to open, pushing his fingers into your mouth. “Here, taste yourself.”

He watched, desire in his eyes as you licked them clean, your cheeks hollowing. He slipped them out from your bruised lips and pressed another kiss to the top of your head.

When you lifted your still-trembling legs to get off his lap, he furrowed his brows, pouting.

“You’re leaving? I was just about to tell you about prions.”

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#criminal minds #spencer reid #fluff #spencer reid smut #spencer reid x reader #smut

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covid-safer-hotties · 6 months ago

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Also preserved in our archive (Daily updates!)

BRONX, N.Y., Nov. 1, 2024 /PRNewswire/ -- A new study led by researchers at Albert Einstein College of Medicine involving more than 200,000 adults found that the COVID-19 pandemic caused a 29% increase in risk for developing dyslipidemia, a condition involving abnormal lipid (fat) levels in the blood. Seniors and people with type 2 diabetes were even more strongly affected, experiencing an approximately two-fold increased risk for developing dyslipidemia, which is a major risk factor for cardiovascular diseases such as heart attack and stroke. The research was published today in the print edition of The Journal of Clinical Investigation.

"Given the extent of the pandemic, this increase in dyslipidemia risk is a cause for concern around the world," said study leader Gaetano Santulli, M.D., Ph.D., associate professor of medicine and of molecular pharmacology at Einstein. "Based on our findings, we would advise people to have their lipid levels monitored regularly and to consult with their healthcare providers about ways to treat dyslipidemia if detected, especially elderly individuals and patients with diabetes." He noted that this advice would apply to all adults, not just those formally diagnosed with COVID-19, considering that many people have been infected without realizing it.

To put these findings into context, it has been estimated that 53% of U.S. adults had dyslipidemia before the pandemic; a 29% increase in dyslipidemia incidence due to COVID-19 would mean that 68% of Americans may now be at risk for having lipid abnormalities.

In two previous studies, Dr. Santulli and his team found that COVID-19 raised the incidence of new cases of hypertension and type 2 diabetes. "In those analyses, we demonstrated that the risk of developing these disorders was still high three years after the pandemic; moreover, we noticed a suspicious increase in total cholesterol levels, which warranted a closer look," said Dr. Santulli. In the new study, the researchers first determined the incidence of dyslipidemia in a group of more than 200,000 adults living in Naples, Italy during the three years prior to start of the pandemic (2017-2019). They then assessed the incidence of dyslipidemia in the same group during the three-year COVID-19 period (2020-2022), excluding from the analysis those people earlier diagnosed with dyslipidemia or who had previously been taking lipid-lowering medications.

The investigators found that COVID-19 raised the risk for developing dyslipidemia in the entire study group by an average of 29%. The increase was even higher among people over age 65 and those with chronic conditions, particularly diabetes and obesity, cardiovascular disease, chronic obstructive pulmonary disease, and hypertension. The findings are the most definitive to date because other studies—most of them linking COVID-19 with modestly increased risks for blood-lipid problems—used as control groups different populations or people thought to have gone through the pandemic without becoming infected. However, significant numbers of people classified as "COVID-free" actually developed the disease but were either never tested or didn't seek medical care.

"Our study did not attempt to determine whether participants had tested positive for COVID-19," Dr. Santulli said. "Instead, because we had been following this group for many years prior to the pandemic, we were able to measure COVID's overall impact on the population by simply comparing levels of dyslipidemia in the same group before and after the pandemic. Any increase in dyslipidemia incidence would almost certainly have to be the result of COVID-19."

How COVID-19 might have increased the incidence of dyslipidemia remains unclear. One possible explanation is a finding Dr. Santulli made in an earlier study: that SARS-CoV-2 (the virus that causes COVID) disrupts the function of endothelial cells, which line the inside of blood vessels throughout the body and play a critical role in regulating blood lipids.

A separate study found that COVID-19 is a powerful risk factor for heart attacks and strokes for as long as nearly three years after an infection. "This investigation, published online a month after ours, essentially confirms our observations in this study, since dyslipidemia is a major contributor to cardiovascular disease," said Dr. Santulli. "It also suggests that tackling dyslipidemia should reduce the risk of cardiovascular disease in those who have had COVID."

The researchers are now studying the effects of COVID-19 on cardiovascular-kidney-metabolic (CKM) syndrome, a recently described condition involving four connected medical problems—heart disease, kidney disease, diabetes, and obesity—all of which involve endothelial dysfunction.

The study is titled "A six-year study in a real-world population reveals an increased incidence of dyslipidemia during COVID-19". The other contributors include: Stanislovas S. Jankauskas, Ph.D., and Fahimeh Varzideh, Ph.D., both at Einstein, Pasquale Mone, M.D., Ph.D., at Einstein and Molise University, Campobasso, Italy, Valentina Trimarco, Raffaele Izzo, Maria Virginia Manzi, Maria Lembo, Paola Gallo, Giovanni Esposito, and Francesco Rozza, at the Federico II University of Naples, Italy, Roberto Piccinocchi, at the Vanvitelli Hospital, Naples, Italy, Carmine Morisco and Bruno Trimarco, at the International Translational Research and Medical Education (ITME) Consortium and Federico II University of Naples, Gaetano Piccinocchi, at the Italian Society of General Medicine, Mario Fordellone and Giuseppe Signoriello, at the Campania University, Naples, Italy.

Study link: www.jci.org/articles/view/183777

#mask up #covid #pandemic #public health #wear a mask #covid 19 #wear a respirator #still coviding #coronavirus #sars cov 2

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evidence-based-activism · 2 months ago

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Sorry to drop this at your feet but I'm too busy trying to pop my eyes back in after rolling them so hard

https://www.tumblr.com/certifiedsexed/771878797272285184/replying-to-this-for-educational-reasons-not-for?source=share

Don't be sorry! This one was interesting to research.

To start out with, I am thoroughly amazed that this person simultaneously believes that biological sex is irrelevant and that being transgender is relevant. If there are no differences between male and female people, then "transitioning" between the two would not be possible. You cannot "transition" between two identical states.

It's also illogical for them to simultaneously believe there are no consistent differences between the sexes, but we also still have to "diversify testing". If the sexes were not dimorphic then all variation in human traits would necessarily be represented within that single sex; therefore, including both sexes in testing would be redundant. This is obviously not the case; that is, women are systemically disadvantaged by male-default bias both in and beyond medical settings [51]. This is because the sexes are dimorphic.

Now, in no particular order, here is a small sample of work demonstrating sexual dimorphism in humans:

Immune System

Men have higher rates of infectious diseases, and women higher rates of autoimmune disorders, suggesting a difference in the degree or type of activity in the immune system. [1]

Women have higher numbers of circulating natural killer cells, a critical component of the immune system. [1]

Differences in sex hormones alter the expression of various immune cells. [1]

Women have a greater response – and greater rate of adverse reactions – to vaccinations than men. [1]

Immune cells in men and women exhibit differences in their "transcriptomics", which means there are sex differences in what proteins are expressed in their cells. [1]

The presence of a second X chromosome is linked to greater rates of autoimmune disorders. [1]

Differences in sex hormones vary the expression of genes that influence immune system behavior. [2]

Differences in sex hormones vary the release of cytokines, which influence the expression and behavior of many different immune cells. [3]

"Perinatal exposure to androgens transforms tissues like brain and genitalia permanently." [3, 4]

Men are more prone to cancer. [24]

Overall, there are simply many, many sex differences in the immune system. [24, 25]

Metabolism

Women often have different disease presentations, treatment responses, and adverse drug reactions than men. [1, 5, 6]

Many factors contribute to the sex differences in drug response, including sex differences in: bioavailability, gut enzymes, protein binding, metabolism (as a result of sex differences in many specific protein activities), and a lot more. [6]

Gender differences in pharmacological response begin prior to birth. [6]

There are significant differences in lipid (fat) metabolism between men and women. In particular, women exhibit higher rates of fatty acid metabolism, show greater fatty acid metabolism during fasting, and more. [7, 13, 14]

Women are more sensitive to insulin. [7, 8, 13]

Women are more susceptible to metabolic syndrome in response to corticosteroids. [9]

Excess androgens (testosterone) induce metabolic dysfunction in women, whereas insufficient androgens induce similar metabolic dysfunction in men. [10]

Men and women exhibit differences in selenium metabolism and activity. [11]

Men and women exhibit differences in glutathione metabolism and activity, which is important for many health conditions including neurodegeneration and heart disease. [12]

Women exhibit greater circulating levels of leptin and adiponectin, which are both involved in metabolism. [17]

Women have a greater metabolic rate in their fat tissue. [30]

Cardiovascular System

The heart is larger in men than in women. [15]

Women and men exhibit differences in heart function, particularly over time. [15, 19, 23]

Men have a higher rate of cardiac coronary heart disease and women have a higher susceptibility to stroke and heart failure. [15, 16]

Men and women show differences in the endothelium system, which mediates heart function. [16]

Men and women exhibit different baseline blood biomarker levels, which impacts the diagnostic process for cardiac diseases. [17]

There are sex differences in G-protein-coupled receptors that contribute to differences in cardiac disease. [18]

Sex hormones result in cellular differences in the heart, such as reduced fibrosis in women and greater hypertrophy in men. [19]

"Some transcriptional and epigenetic sex biases established soon after fertilization persist in cardiac lineages, suggesting that early epigenetic events are perpetuated beyond early embryogenesis. Importantly, when sex hormones begin to circulate, they encounter a cardiac genome that is already functionally distinct between the sexes." [20]

Vascular differences mean that women are more susceptible to orthostatic stress than men. [21]

Men exhibit greater sympathetic cardiovascular activity, and women greater parasympathetic cardiovascular activity, contributing to differences in many cardiovascular diseases. [22]

Women are more prone to lymphatic diseases. [35]

Body Composition

Men have a greater lean mass, and women greater fat mass. [26, 28]

Women store more fat subcutaneously (below the skin), while men store it viscerally (around the organs). [7, 14, 26, 30]

Women have a greater risk of osteoporosis. [26]

Differences in body composition are found across cultures. [27]

There is a small sex difference in height, with men being taller. [28]

Girls have smaller skeletons, specifically, smaller vertebrae (spine bones), which increases their risk for some spinal health conditions. [29]

Sex can be determined by the shape of the skeleton, although this must account for population differences. In other words, within a specific population, male and female skeletons can be differentiated. [31]

Men demonstrate greater muscle strength. [36]

Other Organs

There are sex differences in the skin, including hair growth, sweat rate, sebum production, and more, which impact susceptibility to cutaneous diseases. [32]

There are sex differences in the liver, including response to chronic liver diseases. [33]

Women retain better kidney function as they age. [34]

Men have higher levels of testosterone, and women higher levels of estrogens. [36]

Women have smaller lungs, differently shaped ribs, lower absolute measures of resting pulmonary function, and different responses to exercise. [37]

Men and women have different rates of respiratory diseases. [38]

The Face

There are significant sex differences in the human face. [39, 40]

People can accurately identify someone's sex from their face, although men's faces are easier to identify, and women are better at the task. [41, 42, 43]

Early Sexual Dimorphism

Sex differences in gene expression begin during embryogenesis. [44]

All examined tissues show some degree of sex bias in gene expression. [44]

These differences are critical to understanding sex differences in disease throughout the life course. [45]

There are divergent developmental pathways between the male and female placental formation, function, and adaptation, which plays a role in the sexual dimorphism in disease. [46, 47]

There are sex differences in epigenetic patterning in the placenta, which impacts infant outcomes. [48]

Male embryos are more biologically fragile than female embryos. [49]

Sex differences in the mitochondria impact sex differences in many diseases. [50]

It's important to note that it is not necessary for there to be absolutely no overlap between the sexes for any particular characteristic to be considered sexually dimorphic. It is merely necessary for there to be an average difference on the population level. We should also note that, due to the many, many traits in which humans are sexually dimorphic, an individual with an opposite-sex-average trait will typically still be same-sex-typical for the many other traits.

Ultimately, humans are sexually dimorphic. Attempting to deny biological realities helps no one; not women, not trans people, no one.

References under the cut:

Markle, J. G., & Fish, E. N. (2014). SeXX matters in immunity. Trends in immunology, 35(3), 97-104.

Klein, S. L. (2000). The effects of hormones on sex differences in infection: from genes to behavior. Neuroscience & Biobehavioral Reviews, 24(6), 627-638.

Bhatia, A., Sekhon, H. K., & Kaur, G. (2014). Sex hormones and immune dimorphism. The Scientific World Journal, 2014(1), 159150.

Martin, J. T. (2000). Sexual dimorphism in immune function: the role of prenatal exposure to androgens and estrogens. European Journal of Pharmacology, 405(1-3), 251-261.

Valodara, A. M., & SR, K. J. (2019). Sexual dimorphism in drug metabolism and pharmacokinetics. Current Drug Metabolism, 20(14), 1154-1166.

Franconi, F., Brunelleschi, S., Steardo, L., & Cuomo, V. (2007). Gender differences in drug responses. Pharmacological research, 55(2), 81-95.

Mittendorfer, B. (2005). Sexual dimorphism in human lipid metabolism. The Journal of nutrition, 135(4), 681-686.

Magkos, F., Wang, X., & Mittendorfer, B. (2010). Metabolic actions of insulin in men and women. Nutrition, 26(7-8), 686-693.

Savas, M., Muka, T., Wester, V. L., van den Akker, E. L., Visser, J. A., Braunstahl, G. J., ... & van Rossum, E. F. (2017). Associations between systemic and local corticosteroid use with metabolic syndrome and body mass index. The Journal of Clinical Endocrinology & Metabolism, 102(10), 3765-3774.

Schiffer, L., Kempegowda, P., Arlt, W., & O’Reilly, M. W. (2017). Mechanisms in endocrinology: the sexually dimorphic role of androgens in human metabolic disease. European journal of endocrinology, 177(3), R125-R143.

Seale, L. A., Ogawa-Wong, A. N., & Berry, M. J. (2018). Sexual dimorphism in selenium metabolism and selenoproteins. Free Radical Biology and Medicine, 127, 198-205.

Wang, L., Ahn, Y. J., & Asmis, R. (2020). Sexual dimorphism in glutathione metabolism and glutathione-dependent responses. Redox biology, 31, 101410.

Varlamov, O., Bethea, C. L., & Roberts Jr, C. T. (2015). Sex-specific differences in lipid and glucose metabolism. Frontiers in endocrinology, 5, 241.

Bloor, I. D., & Symonds, M. E. (2014). Sexual dimorphism in white and brown adipose tissue with obesity and inflammation. Hormones and behavior, 66(1), 95-103.

Leinwand, L. A. (2003). Sex is a potent modifier of the cardiovascular system. The Journal of clinical investigation, 112(3), 302-307.

Gohar, E. Y., Giachini, F. R., Pollock, D. M., & Tostes, R. C. (2016). Role of the endothelin system in sexual dimorphism in cardiovascular and renal diseases. Life sciences, 159, 20-29.

Lau, E. S., Binek, A., Parker, S. J., Shah, S. H., Zanni, M. V., Van Eyk, J. E., & Ho, J. E. (2022). Sexual dimorphism in cardiovascular biomarkers: clinical and research implications. Circulation research, 130(4), 578-592.

Mouat, M. A., Coleman, J. L., & Smith, N. J. (2018). GPCRs in context: sexual dimorphism in the cardiovascular system. British Journal of Pharmacology, 175(21), 4047-4059.

Ferreira, C., Trindade, F., Ferreira, R., Neves, J. S., Leite-Moreira, A., Amado, F., ... & Nogueira-Ferreira, R. (2022). Sexual dimorphism in cardiac remodeling: the molecular mechanisms ruled by sex hormones in the heart. Journal of Molecular Medicine, 1-23.

Deegan, D. F., Nigam, P., & Engel, N. (2021). Sexual dimorphism of the heart: genetics, epigenetics, and development. Frontiers in cardiovascular medicine, 8, 668252.

Lindenberger, M., & Lanne, T. (2007). Sex-related effects on venous compliance and capillary filtration in the lower limb. American Journal of Physiology-Regulatory, Integrative and Comparative Physiology, 292(2), R852-R859.

Dart, A. M., Du, X. J., & Kingwell, B. A. (2002). Gender, sex hormones and autonomic nervous control of the cardiovascular system. Cardiovascular research, 53(3), 678-687.

Huxley, V. H. (2007). Sex and the cardiovascular system: the intriguing tale of how women and men regulate cardiovascular function differently. Advances in physiology education, 31(1), 17-22.

Jaillon, S., Berthenet, K., & Garlanda, C. (2019). Sexual dimorphism in innate immunity. Clinical reviews in allergy & immunology, 56, 308-321.

McCombe, P. A., & Greer, J. M. (2014). Sexual dimorphism in the immune system. In The Autoimmune Diseases (pp. 319-328). Academic Press.

Wells, J. C. (2007). Sexual dimorphism of body composition. Best practice & research Clinical endocrinology & metabolism, 21(3), 415-430.

Wells, J. C. (2012). Sexual dimorphism in body composition across human populations: associations with climate and proxies for short‐and long‐term energy supply. American Journal of human biology, 24(4), 411-419.

Lassek, W. D., & Gaulin, S. J. (2022). Substantial but misunderstood human sexual dimorphism results mainly from sexual selection on males and natural selection on females. Frontiers in Psychology, 13, 859931.

Gilsanz, V., Wren, T. A., Ponrartana, S., Mora, S., & Rosen, C. J. (2018). Sexual dimorphism and the origins of human spinal health. Endocrine reviews, 39(2), 221-239.

Palmer, B. F., & Clegg, D. J. (2015). The sexual dimorphism of obesity. Molecular and cellular endocrinology, 402, 113-119.

Ubelaker, D. H., & DeGaglia, C. M. (2017). Population variation in skeletal sexual dimorphism. Forensic Science International, 278, 407-e1.

Giacomoni, P. U., Mammone, T., & Teri, M. (2009). Gender-linked differences in human skin. Journal of dermatological science, 55(3), 144-149.

Lefebvre, P., & Staels, B. (2021). Hepatic sexual dimorphism—implications for non-alcoholic fatty liver disease. Nature Reviews Endocrinology, 17(11), 662-670.

Baylis, C. (2009). Sexual dimorphism in the aging kidney: differences in the nitric oxide system. Nature reviews nephrology, 5(7), 384-396.

Trincot, C. E., & Caron, K. M. (2019). Lymphatic function and dysfunction in the context of sex differences. ACS Pharmacology & Translational Science, 2(5), 311-324.

Handelsman, D. J., Hirschberg, A. L., & Bermon, S. (2018). Circulating testosterone as the hormonal basis of sex differences in athletic performance. Endocrine reviews, 39(5), 803-829.

Molgat-Seon, Y., Peters, C. M., & Sheel, A. W. (2018). Sex-differences in the human respiratory system and their impact on resting pulmonary function and the integrative response to exercise. Current Opinion in Physiology, 6, 21-27.

Reddy, K. D., & Oliver, B. G. G. (2023). Sexual dimorphism in chronic respiratory diseases. Cell & Bioscience, 13(1), 47.

Samal, A., Subramani, V., & Marx, D. (2007). Analysis of sexual dimorphism in human face. Journal of Visual Communication and Image Representation, 18(6), 453-463.

Kleisner, K., Tureček, P., Roberts, S. C., Havlíček, J., Valentova, J. V., Akoko, R. M., ... & Saribay, S. A. (2021). How and why patterns of sexual dimorphism in human faces vary across the world. Scientific reports, 11(1), 5978.

González-Álvarez, J., & Sos-Peña, R. (2022). Sex perception from facial structure: Categorization with and without skin texture and color. Vision Research, 201, 108127.

Cellerino, A., Borghetti, D., & Sartucci, F. (2004). Sex differences in face gender recognition in humans. Brain research bulletin, 63(6), 443-449.

Bruce, V., Burton, A. M., Hanna, E., Healey, P., Mason, O., Coombes, A., ... & Linney, A. (1993). Sex discrimination: how do we tell the difference between male and female faces?. perception, 22(2), 131-152.

Rigby, N., & Kulathinal, R. J. (2015). Genetic architecture of sexual dimorphism in humans. Journal of cellular physiology, 230(10), 2304-2310.

Mauvais-Jarvis, F., Merz, N. B., Barnes, P. J., Brinton, R. D., Carrero, J. J., DeMeo, D. L., ... & Suzuki, A. (2020). Sex and gender: modifiers of health, disease, and medicine. The Lancet, 396(10250), 565-582.

Kalisch-Smith, J. I., Simmons, D. G., Dickinson, H., & Moritz, K. M. (2017). Sexual dimorphism in the formation, function and adaptation of the placenta. Placenta, 54, 10-16.

Gabory, A., Roseboom, T. J., Moore, T., Moore, L. G., & Junien, C. (2013). Placental contribution to the origins of sexual dimorphism in health and diseases: sex chromosomes and epigenetics. Biology of sex differences, 4, 1-14.

Martin, E., Smeester, L., Bommarito, P. A., Grace, M. R., Boggess, K., Kuban, K., ... & Fry, R. C. (2017). Sexual epigenetic dimorphism in the human placenta: implications for susceptibility during the prenatal period. Epigenomics, 9(3), 267-278.

Gutiérrez‐Adán, A., Perez‐Crespo, M., Fernandez‐Gonzalez, R., Ramirez, M. A., Moreira, P., Pintado, B., ... & Rizos, D. (2006). Developmental consequences of sexual dimorphism during pre‐implantation embryonic development. Reproduction in Domestic Animals, 41, 54-62.

Ventura-Clapier, R., Moulin, M., Piquereau, J., Lemaire, C., Mericskay, M., Veksler, V., & Garnier, A. (2017). Mitochondria: a central target for sex differences in pathologies. Clinical science, 131(9), 803-822.

Perez, C. C. (2019). Invisible women: Data bias in a world designed for men. Abrams.

#feminism #postmodernism

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macgyvermedical · 2 months ago

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Hey I know this question might not be in your wheelhouse, but I'm hoping that you might be able to at least point be in a direction with good resources.

I'm writing a story with a character who has an unusually high chance of heart attack in spite of their otherwise healthy lifestyle (for plot reasons), and I think I've landed on "rare lipid disorder" as the cause I want to go with, but I'm having trouble finding resources that really explain the various types beyond "it gives you really high ldl cholesterol!". I don't have any real medical training so I was hoping there's some commonly used resouce I just don't know about.

One thing you could search is "familiar hypercholesterolemia". The most common is heterozygous, where a person can live a normal lifespan if they do appropriate lab testing and take appropriate medications (though they may have xanthelasmas (blebs of cholesterol under the skin around the eyes- see picture above) or xanthomas (cholesterol deposits in joints and tendons). This would be caught whenever the person has their first screening blood test for cholesterol.

The less common type is homozygous, where a person has cholesterol so high (LDL of over 600) that they generally do not survive past age 30 unless they get a liver transplant. The xanthelasmas and xanthomas here are present at birth and this would be caught very quickly.

More information here: https://emedicine.medscape.com/article/121298-overview

#whump reference #writing reference #high cholesterol

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adventremnant · 2 months ago

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This one’s a little personal whoops… So I’m trans and I have ana. And I was about to start T but I am now relapsing hard and I’d rather give up T then give up my ED. I know this isn’t actually a real question but nonetheless it is genuine. Do you think choosing my disordered behaviours over my transition makes me invalid or even fake in my gender identity?

Also how does one manipulate blood test results to look like you’re total healthy and can absolutely start T anyways? (I’ve heard people do that to avoid treatment against their will.)

Also thanks for ur content it’s genuinely informative and helpful!

When you start T, they do a CBC, a lipid panel, and check your estrogen and testosterone levels. Continuing, they'll look at your hematocrit, hemoglobin, estrogen, testosterone, and sex hormone binding globulin.

Taking a daily multivitamin, getting decent protein and fiber, and maybe taking a ferrous sulfate supplement (or prioritizing red meats, dark leafy greens, beans, berries, and seafood) if your multivitamin doesn't contain iron should be sufficient, and none of these tests check for malnutrition anyways.

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darkmaga-returns · 3 months ago

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Part I: The Spike Protein and Its Implications

Dr. Trozzi began by dissecting the central role of the spike protein in both SARS-CoV-2 and mRNA vaccines. He described how these vaccines use lipid nanoparticles to deliver modified mRNA into human cells, instructing the body to produce spike proteins. This process, while intended to stimulate immunity, has led to widespread and unintended consequences.

Weaponization of the Spike Protein: Dr. Trozzi revealed that the spike protein encoded in the vaccines has been genetically engineered to include harmful modifications, such as a furin cleavage site, which increases toxicity, and the removal of hemagglutinin esterase, which naturally counteracts clot formation. These modifications make the spike protein more dangerous than its natural counterpart.

Translation Errors and Contaminants: The modified mRNA in these vaccines is prone to translation errors, leading to the production of random protein fragments that can trigger autoimmune diseases. Additionally, independent research has uncovered contaminants, including plasmid DNA fragments and SV40 promoter sequences, which are known to facilitate the integration of foreign genetic material into human cells. These contaminants raise serious concerns about manufacturing quality and long-term safety.

Systemic Damage: Unlike traditional vaccines, which target specific areas of the body, the lipid nanoparticles in mRNA vaccines allow spike proteins to spread to critical organs, including the brain, heart, ovaries, and testes. This widespread distribution amplifies the potential for harm, contributing to conditions such as myocarditis, reproductive health issues, and neurological disorders.

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biologist4ever · 7 months ago

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Lipid rafts are specialized microdomains within the plasma membrane, rich in cholesterol and sphingolipids, that play a critical role in organizing cellular processes. These dynamic structures act as platforms for signaling molecules, facilitating key functions such as immune responses, cell communication, and membrane trafficking.

Recent research has revealed the importance of lipid rafts in various diseases, including cancer, neurodegenerative disorders, and viral infections. Understanding how lipid rafts organize and modulate cellular activities opens up new possibilities for targeted therapies that could disrupt or harness these microdomains to treat diseases more effectively. As studies on lipid rafts advance, they are proving to be essential components of the plasma membrane's complex architecture and function.

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reality-detective · 2 years ago

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This 👇 was sent to me from a friend. The chat text is my input. Think about it. 👀

White hats are telling their families to turn off cell phones for the Oct 4th EAS test.

From 2-3pm. (There was no indication if the test will be EDT, or will be by time zone.)

Could this be an indication of how the lipid nano particles are planned to be activated using three 1" bursts of 118hz. 5g may be employed for this activation.

This would release the pathogens inside and induce illness on a massive scale--

My thoughts are that this is how they'll begin the next pandemic and a fear based "vax" campaign because people will be so toxic and start dropping dead within weeks.

It has potential to look like ebola as the chimera making the nanobots (probably from graphene) is composed of ebola staphylococcus, brewers yeast and E. coli.

So it could be pretty gruesome if people develop sudden clotting disorders and start bleeding out all of their orifices. I saw this with the 2004(?) SARS patients who were in total organ failure when working in a long term subacute hospital. People will be so freaked out.

These are dots that I have been trying to connect. But the important thing is -- to get the devices shut off because the potential reality is just TOO REAL. 🤔

I've been using Epson salts for almost a year in a 30' hot soak

almost every other day.

Epson 2-3C

Baking soda 1/2-1 C

Borax 1/4-1/2 C

When I drain the tub the following day there was very fine gray "silt"

on the tub bottom even though I'd activated the jets prior to draining the tub.

Two months ago I decided to try magnesium chloride crystals/flakes 2 C instead of epson (MgSO4).

The silt was black! After about 5-6 soaks it began to clear.. so I think lots of graphene/bots (?) had been sucked out with the ionization of the water.

Simultaneously, we'd just started using the fenbendazole 222mg daily to kill the bot producing chimera.

Something is working as my skin no longer burns with the solar flares. Praise god! 🙏🏽

We gotta get this information out there. The potential that the EBS could trigger the next death round and jab campaign is just too real.

Yes... It's theory.

But it's a logical theory. 🤔

#pay attention #educate yourselves #educate yourself #knowledge is power #reeducate yourself #reeducate yourselves #think for yourselves #think about it #think for yourself #do your homework #do your own research #do some research #ask yourself questions #question everything #connect the dots

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breezingby · 9 months ago

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Potential Health Benefits

Honey has many potential health benefits, including: (here's just a Few)…

Anti-inflammatory

Honey contains an enzyme called catalase that can help relieve minor inflammation. It can be used topically to treat burns and wounds, and orally to treat coughs and sore throats.

Antibacterial and antiviral

Honey can help fight infections caused by bacteria, viruses, and fungi. It can also be used to treat acne by dabbing a small amount onto pimples to reduce swelling and fight bacteria.

Antioxidant

Honey contains antioxidants like phenolic acids and flavonoids, which may help improve cholesterol levels and decrease the risk of heart disease. Honey can also help prevent and treat degenerative diseases.

Studies suggest that honey might offer antidepressant, anticonvulsant and anti-anxiety benefits. In some studies, honey has been shown to help prevent memory disorders. Wound care. Topical use of medical-grade honey has been shown to promote wound healing, particularly in burns.

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Cinnamon has many potential health benefits, including:

Blood sugar

Cinnamon may help lower blood sugar levels, which can be beneficial for people with type 2 diabetes. It may also improve insulin sensitivity and glucose tolerance factor (GTf), which can help with weight loss.

In addition to being an antioxidant, anti-inflammatory, antidiabetic, antimicrobial, anticancer, lipid-lowering, and cardiovascular-disease-lowering compound, cinnamon has also been reported to have activities against neurological disorders, such as Parkinson's and Alzheimer's diseases.

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Ginger has many health benefits, including:

Digestion: Gingerol, a natural compound in ginger, can help with digestion, nausea, and vomiting from motion sickness, pregnancy, and cancer chemotherapy. It can also help with mild stomach upset.

Anti-inflammatory: Ginger contains over 400 natural compounds, some of which are anti-inflammatory. It can help with bloating, gas, and reducing inflammation.

Pain relief: Ginger can help with osteoarthritis pain, menstrual cramps, and sore muscles.

Blood sugar: Ginger can help improve blood sugar regulation and lower blood sugar.

Heart health: Ginger can help lower blood pressure and cholesterol, which can help prevent heart disease. It can also improve circulation and promote the breakdown of fats in the bloodstream.

Immune system: Ginger can help strengthen the immune system and fight germs.

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Turmeric ~ In addition to these conditions, research studies have shown some possible benefits of turmeric for:

Inflammation

Degenerative eye conditions

Metabolic syndrome

Arthritis

Hyperlipidemia (cholesterol in the blood)

Anxiety

Muscle soreness after exercise

Kidney health

In India, it was traditionally used for disorders of the skin, upper respiratory tract, joints, and digestive system. Today, turmeric is promoted as a dietary supplement for a variety of conditions, including arthritis, digestive disorders, respiratory infections, allergies, liver disease, depression, and many others.

.......

Chili peppers contain many compounds that may have health benefits, including:

Capsaicin The chemical that gives chili peppers their heat, capsaicin may help with:

Pain relief: Chili peppers can help with headaches, migraines, and joint pain. You can apply chili peppers directly to the skin to reduce the amount of a chemical that sends pain signals to the brain.

Metabolism: Capsaicin can increase your metabolic rate, which can help you burn more calories and eat fewer unhealthy foods. Some studies have also shown that capsaicin can reduce appetite and increase fat burning, which may help with weight loss.

Inflammation: Capsaicin is one of the most studied natural ingredients for its anti-inflammatory properties.

Digestion: Capsaicin can help clear phlegm and congestion, and may promote a healthy gut microbiome, which is important for a healthy immune system.

Vitamin A and vitamin C: Chili peppers are a great source of both vitamins, which can help boost your immune system and support eye health. Vitamin C can also help your body absorb iron and promote healthy skin.

Carotenoids: Chili peppers contain carotenoids, which may help protect against cancer. Some research suggests that capsaicin may also contribute to cancer cell death.

Keep in Mind

Take charge of your health—talk with your health care providers about any complementary health approaches you use. Together, you can make shared, well-informed decisions.

Using Dietary Supplements Wisely

Know the Science: How Medications and Supplements Can Interact

Know the Science: How To Make Sense of a Scientific Journal Article

#Health Benefits #Do Your Research

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drforambhuta · 1 year ago

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The Significance of Blood Tests in Addressing Vertigo:

1. Evaluation of Metabolic and Hormonal Factors: Blood tests serve as valuable tools for assessing metabolic parameters like blood glucose and electrolyte balance, shedding light on hormonal imbalances, particularly related to thyroid dysfunction, which contribute significantly to vertigo. Incorporating cutting-edge technologies, such as continuous glucose monitoring, holds potential for real-time insights into metabolic influences on vertigo. Additionally, examining hormonal fluctuations in various life stages, such as menopause, offers a nuanced understanding of vertigo triggers.

2. Understanding Anemia and Blood Disorders: The role of blood in oxygen transport is crucial for preventing vertigo, and conditions like anemia disrupt this process. A comprehensive examination, including a complete blood count (CBC), proves essential in detecting anemia and various blood disorders, providing insights into potential contributors to vertigo. Combining advanced imaging techniques like functional magnetic resonance imaging (fMRI) with blood tests offers a detailed understanding of physiological changes associated with anemia-induced vertigo. Exploring nutritional deficiencies, such as vitamin B12 and iron, enhances the comprehension and management of anemia-related vertigo.

3. Detection of Infections: Blood tests measuring parameters like white blood cell count and C-reactive protein (CRP) offer crucial insights into infections, guiding clinicians toward targeted treatment strategies addressing both the infection and associated vertigo. Advanced molecular diagnostics, including polymerase chain reaction (PCR) testing, enhance precision in identifying infectious causes of vertigo. Investigating chronic infections and their potential link to persistent vertigo symptoms adds depth to our understanding.

4. Assessment of Lipid Profile: Abnormal lipid levels, particularly high cholesterol, may compromise blood flow to the inner ear, contributing to vertigo. Lipid profile tests are essential in evaluating the cardiovascular health of vertigo patients, emphasizing the interconnectedness of systemic health and vestibular function. Exploring the impact of lifestyle factors on lipid metabolism broadens intervention possibilities for lipid-related vertigo.

5. Unraveling Autoimmune Contributions: Blood tests detecting antibodies associated with autoimmune conditions play a pivotal role in understanding autoimmune-induced vertigo, particularly in conditions like rheumatoid arthritis or lupus. Exploring immunomodulatory therapies and personalized treatment plans based on autoimmune markers provides insights into managing autoimmune-induced vertigo. Investigating the relationship between autoimmune vertigo and comorbidities adds complexity to our understanding.

6. Monitoring Drug Levels: Regular blood tests for drug levels ensure optimal therapeutic benefits without adverse effects, addressing the delicate balance between pharmaceutical management and vestibular health. Advances in pharmacogenomics hold promise in tailoring drug regimens based on individual genetic profiles to minimize medication-induced vertigo. Additionally, exploring the long-term effects of chronic medication use on vestibular function offers a comprehensive view of medication-related vertigo.

Doctors recommend undergoing full body health checkups, including blood tests and other essential tests, at regular intervals to detect and manage conditions such as vertigo early.

#blood tests #blood disorders #metabolic disorders #infections #autoimmune conditions #lipid profile #full body checkups #full body health checkups #regular full body checkups #regular health checkups #anemia

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bpod-bpod · 1 year ago

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Vital for Immunity

Importance of peroxisomes – organelles inside cells that are involved in lipid metabolism – for immune cell development and immune responses revealed by defects seen in a peroxisome-deficient mouse model of the congenital disorder Zellweger disease

Read the published research article here

Image from work by Brendon D. Parsons and Daniel Medina-Luna, and colleagues

University of Alberta, Department of Laboratory Medicine and Pathology, Edmonton, AB, Canada

Image originally published with a Creative Commons Attribution – NonCommercial – NoDerivs (CC BY-NC-ND 4.0)

Published in Cell Reports, February 2024

You can also follow BPoD on Instagram, Twitter and Facebook

#science #biomedicine #immunofluorescence #biology #congenital disorders #zellweger disease #peroxisomes #organelles #cells #immunity

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teachingrounds · 27 days ago

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Q. What is a "cherry red spot" on ophthalmological exam, and what is its significance for pediatric patients?

A. A cherry-red spot reflects the blood flow at the foveola, where the inner retinal layer thins, surrounded by macula that is either lightened by lack of block flow (e.g. retinal ischemia) or opacified by the accumulation of some substance, such as in lipid storage disorders such as Tay-Sachs Disease (gangliosides) [see image] or Niemann-Pick Disease (sphingomyelin) that may be diagnosed in infancy or very early childhood. This week we'll discuss acid sphingomyelinase deficiency.

Pro-Tip: The foveola is only "cherry red" in individuals of light complexion. In darker complexions the spot may be brown or black. This condition might better be described as a "perifoveal white patch." It fades as the neurons atrophy.

Ospina LH, Lyons CJ, McCormick AQ. "Cherry-red spot" or "perifoveal white patch"? Can J Ophthalmol. 2005 Oct;40(5):609-10.

Source: Tripathy K, Patel BC. Cherry Red Spot. [Updated 2023 Aug 25], StatPearls.

#TeachingRounds #FOAMed #pediatrics #genetics #ophtho #ophthalmology #rare disease #physical exam

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prakashdiabetesspeciality · 1 month ago

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Top Diabetes Specialist Doctor in Patna: Expert Profiles, Real-Life Success Stories, and Local Insights

Diabetes is increasing at a rapid rate in India as a whole. Patna is no different. As people become more urbanized and lifestyles change, people are increasingly fighting this chronic disease on a day-to-day basis. Some of the best Diabetes Specialist doctors in Patna are not only fighting diabetes but also striving to develop advanced treatment and individualized care. This article highlights some of the top diabetes specialist doctors in Patna. It talks about their features and treatments, including offering case studies and real-life statistics that put their impact into perspective.

Growing Challenges of Diabetes in Patna

Recent studies indicate that the incidence of diabetes is increasing across India. Studies indicate that approximately 9-10% of the adult population has diabetes. In urban areas like Patna, the number is even higher. Due to lifestyle and dietary factors, a Times of India study found that more than 11 per cent of people in their 20s in the region suffer from diabetes. These figures underscore the imperative for prevention and proper management in our society.

The Patna Regional Health System is making efforts to combat these issues. Advanced diagnostic facilities and telemedicine facilities in modern hospitals enable patients to better control their blood sugar levels. The top diabetes specialist physicians in Patna are doing wonders on a day-to-day basis by combining the latest treatments, diet counselling, and patient education.

Top Diabetes Specialist Doctor in Patna

Here, we will introduce you to some of the top diabetes specialist physicians in Patna, along with their speciality, treatment, and success stories.

1. Prakash Diabetes Hospital

Qualifications and Background:

Dr. Prakash is the Director of the Diabetes Department of the clinic. This advanced centre boasts state-of-the-art technology, state-of-the-art treatments, and low costs. The hospital has a friendly environment and is staffed with dedicated doctors, along with well-trained technicians and nurses.

Treatment:

Dr Prakash provides holistic management of Diabetes and risk factor. By the treatment, his blood glucose levels came down to glycaemic target. And within 3 months of therapy HbA1C came down to HbA1C below 7. Apart from Diabetes, it is very important to control the blood pressure, lipids and anaemia. Monitoring of blood glucose by glucometer will benefit the patient and better adherence with diet and exercise will be achieved

Case studies and real-life statistics:

A 45-year-old executive presented with poorly controlled glycaemia and an initial presentation of neurosis. With the initiation of Dr Prakash's regimen of systematic management with individualised dietary counselling, high-technology gadgetry, and regular follow-up, his HbA1c level decreased from 9.5% to 7.2% in six months. These findings demonstrate the effectiveness of the centre in lowering complication rates and quality of life among patients. Local data also show that better control of blood sugar levels in specialist clinics can reduce cardiovascular complication risk by up to 30%.

2. Dr. Sajjad Ahsan - Diabetes, Thyroid, and Endocrinology Clinic

Qualifications and History:

Dr Sajjad Ahsan is one of the top diabetes specialist doctors in Patna and a respected endocrinologist. With over 12 years of experience, he runs a hospital in Anizabad, Patna, treating diabetes and related endocrine disorders. Dr Ahsan is known for his patient-centred approach and ability to explain complex treatment options in simple, easy-to-understand terms.

Treatment:

Dr. Azan focuses on a holistic approach to diabetes management. His treatment strategy includes close monitoring of blood sugar levels. He prescribes individualized medications and advises on lifestyle changes. He also emphasises the importance of early detection of complications such as retinopathy or optic neuropathy. To ensure timely intervention, his clinic utilises both conventional medicine and novel therapies such as DPP-4 and SGLT2 antagonists to provide comprehensive healthcare to patients.

Case Studies and Real-Life Statistics:

One patient was a 52-year-old teacher diagnosed with type 2 diabetes and thyroid disease. After a detailed evaluation and a comprehensive treatment regimen of oral hypoglycemic medications and lifestyle changes, her fasting blood sugar levels returned to normal. Her overall health improved significantly. Dr. Azzan's approach has contributed to the clinic's overall statistics. Over 70 per cent of patients achieved targeted blood sugar control within a year of starting treatment. These numbers are encouraging given the complexity of managing complications.

3. Dr. Amit Kumar - Diabetologist

Qualifications and History:

Dr Amit Kumar is a 25-year-old top diabetes specialist doctor in Patna, practising on West Boring Canal Road, Patna. He is known for his extensive clinical experience and dedication to patient education. With his many years of medical experience, he has helped numerous patients improve their diabetes from difficult to easier to manage under expert guidance.

Treatment:

Dr. Amit Kumar combines traditional treatment methods with advanced technologies. He is known for his personalised treatment protocols. This may include detailed blood sugar testing and insulin adjustments. Oral hypoglycemic medications and dietary advice. Dr Kumar also encourages patients to follow up regularly and emphasises the role of self-management in diabetes control. His clinic has introduced new digital tools for continuous remote monitoring of patient's health, engaging them to actively participate in their treatment plans.

Case Studies and Real-Life Statistics:

One interesting case involved a 60-year-old patient with chronic type 2 diabetes and multiple comorbidities, including hypertension and hyperlipidemia. Under the care of Dr. Amit Kumar, the patient's medication regimen was further improved. This resulted in a significant reduction in type 2 glycated haemoglobin (HbA₁C) levels from 10.2% to 7.8% over eight months. These therapies not only enhance patients' quality of life but also decrease the risk of complications due to diabetes. Dr Kumar reports that he observed about 65 per cent of patients showing substantial improvement in blood sugar levels after one year of starting targeted therapy.

4. Dr. Vijan Charan – Shrestha Health Clinic

Qualifications and History:

Dr Bijan Charan is an expert general practitioner in diabetes. He is based at Shrestha Health Clinic in Sagunamur, Patna, and has over 20 years of experience. He is recognized for his compassionate approach and realistic treatment protocols in the medical as well as psychological areas of diabetes.

Treatment:

Dr. Charan's approach is a combination of tight clinical management and education of the patient. Dr. Charan keeps his patients' blood glucose closely monitored and employs a combination of the latest medications and lifestyle modifications. Dr Charan also places special emphasis on counselling and behaviour change to help patients deal with the daily challenges of chronic disease self-management.

Case Studies and Real-Life Statistics:

There is a touching case study of a young boy who was diagnosed with type 2 diabetes and struggled with the disease from the onset. Under the care of Dr Sharan, through emphasis on major dietary modifications, exercise, and stress management, the patient not only lowered his HbA1c from 8.8% to 6.9% but also his energy levels and general quality of life improved dramatically. Hospital records indicate that over 60 per cent of patients treated at Shrestha Health Hospital recorded equivalent improvements within six months of starting treatment.

The Way Forward: Technological Advances and New Therapies

One thing the top diabetes specialist doctors in Patna have in common is their willingness to embrace technological advancements. They offer a continuous glucose monitoring app on smartphones to track food and activity, and a telemedicine platform that allows patients to receive expert advice without long waits. These innovations not only help with precise blood sugar control but also provide patients with real-time information to make better decisions about their health.

Moreover, new drug combinations, such as GLP-1 receptor agonists and SGLT2 blockers, are revolutionizing treatment options. These medications also have the added benefit of aiding weight loss and protecting the cardiovascular system. This is especially important given the high rates of comorbidities in this region.

#Diabetes care specialist in patna #Diabetes Speciality Hospital in patna #Best Diabetologists in Patna #Diabetes Specialist Doctors in Patna #Best Doctor for Sugar in Patna #Diabetologist in Patna #Top Diabetes specialist doctor in Patna

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